Scientists from Augusta University found chronic neuropsychiatric symptoms following COVID-19 infection, although self-reported symptoms are not linked to dysfunction.
In the study, they recruited COVID-19-positive adult patients from inpatient and outpatient settings in Georgia.
They examined the severity and chronicity of neurologic findings observed during the acute phase of infection during a five-year period following infection.
The study reported preliminary results from the first 200 patients who were 125 days past having a positive COVID-19 test, on average.
The researchers found that the most reported symptom in the study cohort was fatigue (68.5 percent). Overall, 30 percent of participants had hyposmia and 30 percent had hypogeusia.
There was no link between self-reported neurologic dysfunction and dysfunction on neurologic testing.
There were associations observed for self-reported symptoms and comorbidities with depression and anxiety.
The team found that compared with people who did not have COVID-19 infections, people who had COVID19 performed worse on cognitive testing.
Hypogeusia is a reduced ability to taste things (to taste sweet, sour, bitter, or salty substances). The complete lack of taste is referred to as ageusia.
Causes of hypogeusia include the chemotherapy drug bleomycin, an antitumor antibiotic, Bell’s Palsy, and zinc deficiency among others.
For more information about COVID, please see recent studies about the cause of severe inflammation in COVID-19, and results showing new way to prevent a broad range of COVID-19 variants.
The research was published in Brain, Behavior & Immunity: Health and conducted by Alex K. Chen et al.
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